These infections remain a significant cause of morbidity and mortality worldwide, being responsible for significant healthcare costs. Surgical site infections (SSI) are the most common type of medical-related infection in developing countries, and the second most frequent type in Europe and the United States. Pre-surgical skin asepsis protocols with povidone-iodine or chlorhexidine showed similar efficacy in the elimination of methicillin resistant bacteria and preventing surgical site infections in dogs undergoing surgery. False positives were mainly other staphylococci species, as well as Enterobacteriaceae. Also, the logarithmic reduction of the bacterial quantification from pre- and post-asepsis time, was not statistically different for povidone-iodine (6.51 ± 1.94 log10) and chlorhexidine (6.46 ± 2.62 log10) protocol.įrom the 39% pre-asepsis swabs which showed bacterial growth in MRSA modified chromogenic agar medium, only one isolate was identified as Staphylococcus aureus and one as S. In only 9% of the cases a significant bacterial logarithmic reduction was not observed, indicating possible resistance to these agents. Most samples collected at the post-asepsis did not present bacterial growth, both for the animals subjected to the povidone-iodine (74%) or to the chlorhexidine (70%) protocols. For each dog, two skin swab samples were collected at pre-asepsis and post-asepsis, for bacterial quantification by conventional techniques and isolation of methicillin-resistant species. A total of 46 animals were randomly assigned for an asepsis protocol with an aqueous solution of 7.5% povidone-iodine or with an alcoholic solution of 2% chlorhexidine. The aim of this study was to evaluate the effectiveness of two pre-surgical skin asepsis protocols in dogs. However, in veterinary medicine there is no agreement on which biocide is the most effective. Pre-surgical asepsis of the skin is one of the preventive measures performed to reduce SSI incidence and also antibiotic resistance dissemination. Most of surgical site infections (SSI) are caused by commensal and pathogenic agents from the patient’s microbiota, which may include antibiotic resistant strains.
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